Cheng Wang
department
Nephrology
Introduction

Cheng Wang M.D. & Ph.D.


Titles

Associate Professor

Division of nephrology, Department of medicine, 3rd Hospital of Sun Yat-sen University

 

Research Interests

·Blood pressure pattern in CKD

·Microvascular function in CKD

·Podocyte injury and its mechanism

 

Education and Experience

Education

 M.B (Medical Bachelor, M.D equivalent) 2001 Sun Yat-sen University, Medical College, Guangzhou, P.R. China

 M.S 2003 Sun Yat-sen University, College of Graduate Studies, Guangzhou, P.R. China

Ph.D 2008 Sun Yat-sen University, College of Graduate Studies, Guangzhou, P.R. China

Postdoctor fellowship 2012, Division of nephrology and hypertension, Georgetown University, USA

 

Experience

Resident (2003-2005), Division of nephrology, Department of Medicine, 3rd hospital of Sun Yat-sen University, Guangzhou, P.R. China

Attending physician (2006-2009), Division of nephrology, Department of Medicine, 3rd hospital of Sun Yat-sen University, Guangzhou, P.R. China

Associate professor (2009- ), Division of nephrology, Department of Medicine, 3rd hospital of Sun Yat-sen University, Guangzhou, P.R. China

 

Honors and Awards

Dr. Wang is a young member of the standing committee of Guangdong Medical AssociationKidney Disease Branch. he is honored as distinguished younger doctor in 3rd hosptial.

 

Publications

1. Wang Cheng, Luo Zaiming, Donald Kohan, Anton Wellstein, Pedro A. Jose, William J. Welch,, Christopher S. Wilcox, Wang Dan. Thromboxane prostanoid receptors enhance contractions, endothelin-1 and oxidative stress in microvessels from mice with CKD. Hypertension 2015,65: 1055-63

2. Wang C, Zhang J, Deng W, Gong W, Liu X, Ye Z, Peng H, Lou T. Nighttime Systolic Blood-Pressure Load Is Correlated with Target-Organ Damage Independent of Ambulatory Blood-Pressure Level in Patients with Non-Diabetic Chronic Kidney Disease. PLoS One 2015;10(7):e0131546. doi: 10.1371

3. Wang C, Deng W, Gong WY, Zhang J, Tang H, Peng H, Zhang QZ, Ye ZC, Lou T. High prevalence of isolated nocturnal hypertension in Chinese patients with chronic kidney disease. J Am Heart Assoc 2015 ;4(6):e002025. doi: 10.1161

4. Wang C, Deng W, Gong WY, Zhang J, Zhang QZ, Ye ZC, Lou T. Nocturnal Hypertension Correlates Better With Target Organ Damage in Patients With Chronic Kidney Disease than a Nondipping Pattern. J Clin Hypertens (Greenwich) 2015. doi: 10.1111/jch.12589

5.  Wang C, Gong WY, Zhang J, Peng H, Tang H, Liu X, Ye ZC, Lou T. Disparate assessment of clinic blood pressure and ambulatory blood pressure in differently aged patients with chronic kidney disease. Int J Cardiol  2015, 183:54-62

6. Wang C , Li CC, Peng H, Ye Z, Zhang J, Lou tq. Activation of the Nrf2–ARE pathway attenuates hyperglycemia-mediated injuries in podocytes. Cell Physiol Biochem 2014,34:891-902

7. Wang CLi CC, Gong WY, Lou TQ. New urinary biomarkers for diabetic kidney disease. Biomarker Research 1:9 doi:10.1186/2050-7771-1-9,2013

8. Wang C, Liu X, Ke Z, Tang Y, Li CC, Li CM, Ye Z, Zhang J, Lou T. Mesangial medium from IgA nephropathy patients induces podocyte epithelial-to-mesenchymal transition through activation of the phosphatidyl inositol-3-kinase/Akt signaling pathway. Cell Physiol Biochem 29(5-6):743-752, 2012

9. Wang C, Zhang J, Liu X, Li CC, Ye ZC, Peng H, Chen Z, Lou T. Effect of valsartan with bedtime dosing on chronic kidney disease patients with nondipping blood pressure pattern. J Clin Hypertens (Greenwich) 15(1):48-54, 2013

10. Wang C, Zhang J, Liu X, Li C, Ye Z, Peng H, Chen Z, Lou T. Reversed dipper blood-pressure pattern is closely related to severe renal and cardiovascular damage in patients with chronic kidney disease. PLOS one 8(2): 1-9, 2013

11. Wang C, Liu X, Tang Y, Peng H, Ye ZC, Zhang J, Tang H, Lou T. Medium from mesangial cells incubated with aggregated IgA1 from IgA nephropathy patients reduces podocyte adhesion through activation of the renin angiotensin system. Swiss Med Wkly. 141:w13304. doi: 10.4414/smw.2011.13304, 2011

12. Wang C, Liu X, Peng H, Tang Y, Tang H, Chen Z, Lou T, Zhang H. Mesangial cells stimulated by immunoglobin A1 from IgA nephropathy upregulates transforming growth factor-beta1 synthesis in podocytes via renin-angiotensin system activation. Arch Med Res 41(4):255-60, 2010

13. Wang C, Liu X, Ye Z,Zhang J, Tang H, Chen Z, Zhang H, Lou T. Mesangial medium with IgA1 from IgA nephropathy inhibit nephrin expression in podocytes Eur J Clin Invest 39(7):561-7 2009

14. Wang C, Ye Z, Peng H, Tang H, Liu X, Chen Z, Yu X, Lou T. Effect of aggregated IgA1 from IgA nephropathy patients on nephrin expression in podocytes. Nephrology (carlton) 14(2):213-8, 2009

 

Contact

Division of Nephrology, Department of Medicine, 3rd Hospital of Sun Yat-sen University

# 600, Tianhe Street, Guangzhou, Guangdong, China, 510630

e-mail: wcgz@medmail.com.cn